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In FM markers of elevated inflammation and immune dysregulation were observed on multiple platforms relative to HC which is very similar to the situation observed in ME/CFS (see earlier sections)

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Insulin-mediated skeletal muscle vasodilation is nitric oxide dependent

proangiogenic factors (i.e., upregulated after wound healing even in the absence of new vessels) (for a review, see, i.e., [126,127,128])